FB2026_02 , released June 18, 2026
FB2026_02 , released June 18, 2026
Aberration: Dmel\Dp(1;f)γ878
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General Information
Symbol
Dp(1;f)γ878
Species
D. melanogaster
Name
FlyBase ID
FBab0024064
Feature type
Also Known As
γ878
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bk2 << p1:bk1

Genetic mapping information
Comments
Comments on Cytology

Terminal euchromatic deletion at position -77 of the Dp(1;f)1187 derivative Dp1187-8-23, where position 0 is the In(1)sc8 inversion breakpoint.

"Terminal" deletion.

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Completely deleted / disrupted
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Complementation Data
 
Molecular Data
 
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations

    Terminal deficiency associated position effect variegation (TDA-PEV) of the y+ gene in Dp(1;f)γ878 is suppressed by Dp(1;f)γ158 ("trans-suppression").

    Dp(1;f)γ878/Dp(1;f)γ158 heterozygotes exhibit a dramatic increase in y+ expression, relative to Dp(1;f)γ878 - nearly complete suppression of terminal deficiency induced PEV. Increased y+ expression is visible in the abdominal cuticle, wings and thoracic and triple row bristles.

    NOT in combination with other aberrations

    Transmitted from meiosis at 29% in nod3/nod+ females and 52% in wild type females.

    y PEV phenotype, partially suppressible by extra Y chromosome.

    Stocks (0)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    Terminal deficiency breakpoint is 55kb from y+ locus.

    Monosome transmission behaviour from both male and female parents is stable demonstrating normal transmission and normal centromere function.

    Synonyms and Secondary IDs (2)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
      References (6)