Abnormal grooming behavior.

At 25^oC the phenotype is wild type, but at 18^oC the wings are scalloped and females are sterile. Most medial tract axons travel in the lateral tract during wing sensory axon development in discs during metamorphosis. Ectopic sensory neurons are displayed in the wing.

Hemizygotes are viable and fertile but result in the alteration or loss of a normally neuron-specific glycoconjugate which is recognized by anti-HRP antibodies. After heat stress at 37^oC for 5 minutes subsequent banging produced a jittery phenotype not observed in controls. At 18^oC flies born from heterozygous parents have morphological abnormalities: wing formation and assembly of eye facets, females are sterile.

Mutants show alteration or loss of a normally neuron-specific glycoconjugate; staining by anti-HRP antibodies in imaginal and adult neural tissue is eliminated. At 25^oC the mutant flies are viable and fertile; nac/nac females, however are sterile at 18^oC. Under heat stress (37^oC for five minutes) they show abnormal jittery behavior. Developmental abnormalities, including defects in the assembly of the ommatidia and in the formation of the wing, appear at 18^oC in the homozygous offspring of heterozygous parents. Maternal effect embryos show loss of the anti-HRP glycan determinant and are lethal.