Abnormal trap-entry with ethyl acetate as the stimulus. This phenotype is at least partially dominant. Normal response to ethyl acetate, propionic acid or benzaldehyde in a chemosensory jump assay. ERG is abnormal in homozygotes. Giant fibre pathway physiology appears normals.

Abnormal trap-entry kinetics (cf. ota1) with ethyl acetate as stimulant. This phenotype is semidominant, as indicated from similar testing of ota7/+ females. Odor-induced jump responses normal. ERG abnormal. The photoreceptor potential begins anomalously to decay to baseline soon after normal light-on transient appears; then renewed (cornea-negative) depolarization is observed (all of these dynamics observed during 0.5 second stimulus). Finally, a normal light-on transient is observed. The abnormal ERG phenotype, which co-segregates with subnormal trap-entry kinetics, is recessive.