Embryos lacking zygotic capt function do not have defects in the embryonic central nervous system.

In capt germ-line clones the distribution of F-actin appears relatively normal during the very early stages of oogenesis, but becomes highly polarised and dynamic as the egg chamber develops. Ectopic F-actin is first seen at stage 5-6 of oogenesis in a dense structure at the posterior pole of the oocyte. By stages 6-8, ectopic actin filaments appear to shift to the anterior of the oocyte where they are found close to ring canals. F-actin underlying the nurse cell membrane disappear s prematurely at stages 8-9 of oogenesis. At stage 10B, dumping fibres can be seen forming on schedule in the nurse cells of capt clones. Finally in eggs, extensive filamentous actin structures form close to the cortex, and ectopic F-actin is visible in the few embryos that are produced. Yolk often fail to form in the oocyte of capt germ-line clones. Instead abnormal yolk particles accumulate at the nurse cell-oocyte boundary. Moreover in stage 7-9 mutant egg chambers the movements characteristic of cytoplasmic streaming are not seen. Expression of Ecol\lacZ^P\T.T:Khc indicates that microtubule organisation is disrupted in capt germ-line clone oocytes. Microtubules remain polarised but are misaligned. In cases where microtubules polarity appears reversed, this altered polarity is accompanied by a change in morphology of the oocyte, which appears to invade the nurse cell cluster.