94.3% of embryos derived from females carrying P{UAS-cbs-RNAi-UAS} (contains the cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} alleles) expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 18^oC are wild type.

34.7% of embryos derived from females carrying P{UAS-cbs-RNAi-UAS} (contains the cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} alleles) expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 25^oC are wild type, while 40.8% have moderate defects at the syncytial stage, 17.3% have severe defects at the syncytial stage and 7.1% have severe defects at the cellular stage.

Only 3.9% of embryos derived from females carrying P{UAS-cbs-RNAi-UAS} (contains the cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} alleles) expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 29^oC are wild type, while 13.4% have moderate defects at the syncytial stage, 68.5% have severe defects at the syncytial stage and 14.2% have severe defects at the cellular stage.

During prophase in syncytial blastoderm embryos, moderate reductions in cbs protein levels (by expression of cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}) result in centrosomes that vary in size, and acentrosomal nuclei are often present. In syncytial blastoderm embryos in which cbs protein levels are more severely depleted, the frequency of acentrosomal nuclei increases and the nuclei appear to be degraded.

During metaphase in syncytial blastoderm embryos, moderate reductions in cbs protein levels (by expression of cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}) result in aberrant spindle formation, which varies from short, broad spindles to completely collapsed spindles, and the aberrant spindles usually have asymmetric centrosomes.

At the start of cellularisation in embryos in which cbs protein levels are significantly depleted (by expression of cbs^Sym.Scer\UAS and cbs^{a.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}), centrosome maturation is impaired for 20-50% of the nuclei. Often only one centrosome is competent to nucleate astral microtubules. As cellularisation proceeds, the nuclei are poorly aligned at the cortex and many drop from the cortex.

94.3% of embryos derived from females carrying cbs^{dsRNA.Sym.Scer\UAS} expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 18[o]C are wild type.

34.7% of embryos derived from females carrying cbs^{dsRNA.Sym.Scer\UAS} expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 25[o]C are wild type, while 40.8% have moderate defects at the syncytial stage, 17.3% have severe defects at the syncytial stage and 7.1% have severe defects at the cellular stage.

Only 3.9% of embryos derived from females carrying cbs^{dsRNA.Sym.Scer\UAS} expressed under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16} at 29[o]C are wild type, while 13.4% have moderate defects at the syncytial stage, 68.5% have severe defects at the syncytial stage and 14.2% have severe defects at the cellular stage.

During prophase in syncytial blastoderm embryos, moderate reductions in cbs protein levels (by expression of cbs^{dsRNA.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}) result in centrosomes that vary in size, and acentrosomal nuclei are often present. In syncytial blastoderm embryos in which cbs protein levels are more severely depleted, the frequency of acentrosomal nuclei increases and the nuclei appear to be degraded.

During metaphase in syncytial blastoderm embryos, moderate reductions in cbs protein levels (by expression of cbs^{dsRNA.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}) result in aberrant spindle formation, which varies from short, broad spindles to completely collapsed spindles, and the aberrant spindles usually have asymmetric centrosomes.

At the start of cellularisation in embryos in which cbs protein levels are significantly depleted (by expression of cbs^{dsRNA.Sym.Scer\UAS} under the control of Scer\GAL4^{nos.UTR.T:Hsim\VP16}), centrosome maturation is impaired for 20-50% of the nuclei. Often only one centrosome is competent to nucleate astral microtubules. As cellularisation proceeds, the nuclei are poorly aligned at the cortex and many drop from the cortex.