17% of mutant animals die before reaching adulthood, and the surviving adults are flightless, showing severe degeneration of the indirect flight muscles.

Ovarian morphology is severely disrupted in mutant females. Malformed or missing stalks, egg chamber fusions, abnormal germarial structure and aberrant follicle cell sizes and morphologies are seen. Abnormal ring canal morphology, deficient transfer of nurse cell contents to the oocyte and disorganised actin cages are also seen. The phenotypes are variable and increase in severity with age.

Germline stem cells accumulate in aged females and the increase in germline stem cell number is accompanied by an increase in cap cell number.

Mutant egg chambers contain elevated numbers of polar cells, which are not always located at the poles.

Filamentous actin appears normal in the germaria of mutant females.

In ovarioles in which all the follicle cells are homozygous, the mutant phenotypes are similar to those seen in homozygous females. In ovarioles containing homozygous germline cells, stalks form largely normally between the egg chambers and encapsulation occurs normally.