UAS regulatory sequences drive expression of a short inverted repeat.
Embryos from mothers expressing Ipo9HMS00804 under the control of Scer\GAL4VP16.mat.αTub67C show widespread mitotic defects during the earliest embryonic divisions (chromosome bridges, chromosome fragmentation, lack of chromosome condensation and an array of spindle defects).
Expressing Ipo9HMS00804 under the control of Scer\GAL4VP16.nos.UTR leads to spermatogenesis defects, with few spermatids going through individualization.