UASt regulatory sequences drive expression of an inverted repeat.
Expression of OcrlKK101922 under the control of any of these drivers: Scer\GAL4He.PZ, Scer\GAL4HHLT, Scer\GAL4ppl.PU or Scer\GAL4Hand.PU does not cause excessive accumulation of circulating hemocytes in third instar larvae (and thus replicate the phenotype of Ocrl classical loss-of-function alleles), presumably due to low knock-down efficiency.