Df(1)BSC882 was isolated as a FLP recombinase-induced recombination event involving PBac{WH}Atx-1f01201 and P{XP}d11388. The deletion was isolated as a chromosome carrying two copies of the miniwhite marker in progeny of PBac{WH}Atx-1f01201/P{XP}d11388; MKRS, P{hsFLP}86E/+ females crossed to FM7h/Dp(2;Y)G, P{hs-hid}Y males. These females were heat shocked as larvae as described in Parks et al., Nature Genetics 36: 288-292, 2004 (FBrf0175003). This cross and crosses in preceding and succeeding generations maintained the original genetic background of the Exelixis insertion stocks (Thibault et al., Nature Genetics 36: 283-287, 2004; FBrf0175002). The recombination event generated the genetic element P+PBac{XP3.WH3}BSC882 from the segment of PBac{WH}Atx-1f01201 to the left of its FRT site and the segment of P{XP}d11388 to the right of its FRT site. The breakpoints of Df(1)BSC882 predicted from the Release 5 genomic coordinates of the transposable element insertion sites are  X:6718207 ;6909441 and the cytological breakpoints predicted from these coordinates are 6D3;6E4. To confirm the presence of the deficiency, we showed that a 883 base pair fragment spanning the PBac{WH}cmf05774 (FBti0042490) insertion site could not be amplified from Df(1)BSC882/PBac{WH}cmf05774 flies using primers 5’-CCACATAGTTGTCCTTGATCACGG-3’ and 5’-CCGTGTTTGTGTATTGCCTCCGTCAC-3’.