Marker analysis indicates that pre-and post synaptic component of the neuromuscular junction fail to align properly in larvae homozygous for Atg1EY09216.
Atg1EY09216/Atg1[+] is a suppressor of abnormal oxidative stress response | dominant phenotype of pucE69
Heterozygosity for Atg1EY09216 or Atg600096 reverts the resistance to paraquat toxicity seen in pucE69/+ animals.
Identified in a screen for mutants in which apposition of post-and pre-synaptic neuro-muscular junction components fails