Discovered during the characterization and localization of loci within the 95E--96F region.
Adult flies heterozygous for aor1 (In(3R)Ubx7L) and a deficiency for 96A show two separable phenotypes: a recessive loss of function phenotype causing reduction or disappearance of the first abdominal segment and a dominant enhancement of the Ubx transformation of haltere into wing. This dominant effect results from the interaction of lesions in Ubx with the haplo-insufficiency of the region 96A. The addition of an extra dose of the region 96A can partially suppress the Ubx phenotype. The recessive phenotype is independent of mutations at Ubx.
Discovered during the characterization and localization of loci within the 95E--96F region.
Adult flies heterozygous for aor1 (In(3R)Ubx7L) and a deficiency for 96A show two separable phenotypes: a recessive loss of function phenotype causing reduction or disappearance of the first abdominal segment and a dominant enhancement of the Ubx transformation of haltere into wing. This dominant effect results from the interaction of lesions in Ubx with the haplo-insufficiency of the region 96A. The addition of an extra dose of the region 96A can partially suppress the Ubx phenotype. The recessive phenotype is independent of mutations at Ubx.