FB2026_01 , released March 12, 2026
FB2026_01 , released March 12, 2026
Allele: Dmel\Vno1
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General Information
Symbol
Dmel\Vno1
Species
D. melanogaster
Name
FlyBase ID
FBal0018050
Feature type
allele
Associated gene
Vno
Associated Insertion(s)
Carried in Construct
Key Links
Allele class
Mutagen
Nature of the Allele
Allele class
Mutagen
X ray
(Grell, 1959)
Progenitor genotype
Caused by aberration
Tp(3;3)Vno
Cytology
Description
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
 
UniProtKB/Swiss-Prot
    UniProtKB/TrEMBL
      Expression Data
      Reporter Expression
      Additional Information
      Statement
      Reference
       
      Marker for
      Reflects expression of
      Reporter construct used in assay
      Human Disease Associations
      Disease Ontology (DO) Annotations
      Models Based on Experimental Evidence ( 0 )
      Disease
      Evidence
      References
      Modifiers Based on Experimental Evidence ( 0 )
      Disease
      Interaction
      References
      Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
       
      Disease-implicated variant(s)
       
      Phenotypic Data
      Phenotypic Class
      Phenotype Manifest In
      Detailed Description
      Statement
      Reference

      When e1 is combined with Vno1, in which wing veins are truncated or eliminated, ectopic melanin only develops in proximal areas that truncate before reaching the margin.

      (True et al., 1999)

      Heterozygotes have terminal gaps in all longitudinal veins in the following order: LII > LIV > LV > LIII. The density of the chaetae in the wing margin is reduced.

      (Diaz-Benjumea and Garcia-Bellido, 1990)

      Second longitudinal wing vein has a sizeable gap and the fourth wing vein is often broken and sometimes the fifth vein is also affected.

      (Grell, 1959)
      External Data
      Interactions
      Show genetic interaction network for Enhancers & Suppressors
      Phenotypic Class
      Phenotype Manifest In
      Enhanced by
      Statement
      Reference

      Vno1 has phenotype, enhanceable by Egfrt1/Egfrf11

      (Diaz-Benjumea and Garcia-Bellido, 1990)
      Enhancer of
      Statement
      Reference

      Vno1/Vno[+] is an enhancer of phenotype of Egfrt1/Egfrf11

      (Diaz-Benjumea and Garcia-Bellido, 1990)
      Additional Comments
      Genetic Interactions
      Statement
      Reference

      Egfrt1/Egfrf11 Vno1/+ double mutants have a superadditive phenotype. EgfrE3/+ shows a negative interaction in combination with Vno1/+.

      (Diaz-Benjumea and Garcia-Bellido, 1990)
      Xenogenetic Interactions
      Statement
      Reference
      Complementation and Rescue Data
      Comments
      Images (0)
      Mutant
      Wild-type
      Stocks (2)
      Notes on Origin
      Discoverer

      E.H. Grell, March 1956.

      E.H. Grell.

      (Grell, 1959)

      The dominant wing phenotype (represented by Vno1) caused by the Tp(3;3)Vno chromosome may be associated with any of the 93F, 94A or 96F11 breakpoints. It is not caused by haploinsufficiency of the locus because the dominant phenotype can be fully reverted by mutation. The 89E breakpoint of the Tp(3;3)Vno chromosome is associated with a bithorax phenotype (represented by Abd-BVno).

      (Diaz-Benjumea and Garcia-Bellido, 1990)
      External Crossreferences and Linkouts ( 0 )
      Synonyms and Secondary IDs (3)
      Reported As
      Symbol Synonym
      Vno1
      Vno3
      Vnoiab6-Vno
      (Grell, 1959)
      Name Synonyms
      Secondary FlyBase IDs
      • FBal0030378
      References (4)