Clones in the head homozygous for ocotd-XC86 show abnormal morphology in the frons.
Defects in head development and segmental patterning. Abnormal neuropil. Aberrant commissure formation in each segment. The disappearance of neurons labelled by P{A92}45C demonstrates that cell death is restricted to identified neurons associated with the midline of the CNS.
Maternal germline clonal analysis demonstrates there is no maternal effect.
ocotd-XC86 is partially dominant in combination with RpS5a2.