Mutant embryos show normal macrophage dispersal.

84% of ommatidial clusters in homozygous clones in the eye show abnormal arrangement of the R3 and R4 cells, and 67% show misrotation of the cells. Mosaic ommatidia at the borders of mutant clones show a number of polarity defects, including reversed chirality (56%), misrotation (44%) and loss of R3/R4 asymmetry (30%). Wild-type ommatidia adjacent to mosaic ommatidia do not show polarity defects. Mosaic ommatidia with incorrect chirality and/or rotation always contain at least one mutant outer photoreceptor cell (R1-R6). For misoriented ommatidia, R3 is mutant in 49% of cases, R4 is mutant in 36% of cases and R6 is mutant in 26% of cases. A mosaic ommatidium must contain at least five peb⁺ photoreceptor cells to be able to maintain its integrity in the retinal epithelium. Mutant rhabdomeres in mosaic ommatidia fail to extend the normal length of the ommatidial column, indicating that the cells often have shortened cell bodies. Rhabdomere separation is often normal in the apical region, but more basally, rhabdomeres of adjacent cells often fail to separate. Clones in the eye disc show normal levels of apoptosis during late larval and early-to-mid pupal stages.

The salivary glands are in an abnormal position in mutant embryos.

Displays locomotor activity rhythm with an approximately 24h period.