FlyBase curator comment: this entry is used to capture phenotypic information when the particular allele (or allele combination) used by the author could not be determined but the context of the experiment suggests that the phenotype being described is some kind of loss of function.
F-actin bundles in the developing bristles of mutant adults are significantly larger than normal.
Embryos mutant for loss of function mutations show axon guidance defects in ISNb, SNa and the CNS. The majority of defects are manifest as a decrease in repulsion.
The outer Fas2-positive bundle of the longitudinal tracts is discontinuous in plexAunspecified embryos.
PlexAunspecified/plexA[+] is a non-suppressor of visible phenotype of MicalΔPIR.UAS, Scer\GAL4B11-98
PlexAunspecified has larval longitudinal connective phenotype, suppressible by Fas2[+]/Fas2unspecified
PlexAunspecified/plexA[+] is a non-suppressor of macrochaeta phenotype of MicalΔPIR.UAS, Scer\GAL4B11-98
plexAunspecified/+ does not suppress the branched bristle phenotype caused by expression of MicalΔPIR.Scer\UAS under the control of Scer\GAL4B11-98.
The longitudinal tract phenotype is suppressed by one copy of Fas2unspecified.