UAS sequences drive expression of a mutated shg that contains a deletion of residues 1672 to 1696 (mutation affects the β-catenin binding sites).
When shgΔarm.Scer\UAS is driven by Scer\GAL4btl.PS tracheal development appears unaffected.
Scer\GAL4btl.PS/shgΔarm.UAS fails to rescue shg2