Expression of CG4769^GD3851 under the control of Scer\GAL4^{Hand.ΔVM.Switch} (initiated by feeding flies RU486 throughout development and adulthood) results in significant heart dilatation. This phenotype is partially rescued upon treatment with methylene blue.

Adults expressing Cyt-c1^GD3851 under the control of Scer\GAL4^elav.PLu (in the presence of Dcr-2^Scer\UAS.cDa to increase the efficiency of RNAi) do not show a significant defect in avoidance of noxious temperature (46[o]C) compared to control flies.

Expression of CG4769^GD3851 under the control of Scer\GAL4^da.Switch.PT in the presence of 20μ/ml RU486 results in a prolonged developmental phase compared to controls.

Expression of CG4769^GD3851 under the control of Scer\GAL4^da.Switch.PT in the presence of 20μ/ml RU486 does not result in a significant change in body weight in male flies.

Expression of CG4769^GD3851 under the control of Scer\GAL4^da.Switch.PT in the presence of 20μ/ml RU486 does not have a significant effect on adult lifespan.

Expression under the control of Scer\GAL4^Mef2.PR results in late larval lethality.

Expression under the control of Scer\GAL4^Mef2.PR results in grossly normal larval body wall muscles.

Expression under the control of Scer\GAL4^Mef2.PR results in wild-type sarcomeres in the larval body wall muscles.

Expression under the control of Scer\GAL4^pnr-MD237 results in bristle morphology defects on the notum in 70-80% of the Scer\GAL4^pnr-MD237 expression domain.

Expression under the control of Scer\GAL4^pnr-MD237 results in the absence of 30-40% of the Scer\GAL4^pnr-MD237-expressing area of the notum.