The synaptonemal complex assembles with proper timing and morphology in the ovaries of Cap-G^Z1/Cap-G^K4 females. However, there is a clear delay in disassembly of the synaptonemal complex in the mutant ovaries.

Stage 12 and 13 oocytes show a modest increase in the number of prometaphase I configurations with separated chromosomes in Cap-G^Z1/Cap-G^K4 females. Stage 14 oocytes show a dramatic increase in metaphase I defects, most commonly multiple chromosome masses. This phenotype is recessive. Premature sister chromatid separation is not seen, but the X homologs and autosomes do prematurely disjoin from their bivalents.

Embryos derived from Cap-G^Z1/Cap-G^K4 females arrest early, with defects such as polyploid masses.

The condensed chromosomes normally present in the polar body rosette structure of early embryos have aberrant morphology in embryos derived from Cap-G^Z1 females; the chromosomes are hypercondensed.