FB2026_02 , released June 18, 2026
Allele: Dmel\Su(H)EG37
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General Information
Symbol
Dmel\Su(H)EG37
Species
D. melanogaster
Name
FlyBase ID
FBal0244435
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Key Links
Allele class
Nature of the Allele
Allele class
Progenitor genotype
Cytology
Description
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DNA sequence
Protein sequence
 
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Disease-implicated variant(s)
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference
External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhancer of
Statement
Reference

Su(H)[+]/Su(H)EG37 is an enhancer of visible | dominant phenotype of DCTN1-p150Gl-1

Su(H)[+]/Su(H)EG37 is an enhancer of visible phenotype of DCTN1-p150Δ.UAS, Scer\GAL4GMR.PF

Phenotype Manifest In
Enhancer of
Statement
Reference

Su(H)[+]/Su(H)EG37 is an enhancer of eye phenotype of DCTN1-p150Gl-1

Su(H)[+]/Su(H)EG37 is an enhancer of ommatidium phenotype of DCTN1-p150Gl-1

Su(H)[+]/Su(H)EG37 is an enhancer of rhabdomere phenotype of DCTN1-p150Gl-1

Su(H)[+]/Su(H)EG37 is an enhancer of eye phenotype of DCTN1-p150Δ.UAS, Scer\GAL4GMR.PF

Additional Comments
Genetic Interactions
Statement
Reference

Su(H)EG37 dominantly enhances the eye phenotype caused by expression of GlΔ.Scer\UAS under the control of Scer\GAL4GMR.PF.

Su(H)EG37 dominantly enhances the eye phenotype caused by Gl1, resulting in an increase in bristles and an increase in ommatidial fusion and disorganisation. There is increased disruption of accessory cells and rhabdomeres are often fused.

The electrophysiological defects seen in the giant fiber system of Gl1/+ adults are enhanced if they are also heterozygous for Su(H)EG37; the response latencies are significantly increased and the double heterozygous flies show poor following.

Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Comments
Images (0)
Mutant
Wild-type
Stocks (0)
Notes on Origin
Discoverer

It has not been determined whether the lethality of the chromosome is caused by the mutation that modifies the eye phenotype caused by expression of GlΔ.Scer\UAS under the control of Scer\GAL4GMR.PF, so it is possible that the recessive lethality of the chromosome is due to a second-site mutation.

External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (1)
Reported As
Symbol Synonym
Su(H)EG37
Name Synonyms
Secondary FlyBase IDs
    References (1)