UAS regulatory sequences drive expression of Hsap\ADARB1 in which the Ser corresponding to the self-editing site in the orthologous Adar protein has been mutated to Gly.
Hsap\ADARB1G.UAS/Scer\GAL4ChAT.7.4 is a non-suppressor of abnormal locomotor behavior | adult stage phenotype of Adar5G1