The ectopic expression of Gr22b^Scer\UAS.cDa under the control of Scer\GAL4^Gr89a.2 leads to the following electrophysiological response changes of labellar sensilla to bitter tastants, as compared to controls: I-a and I-b sensilla display novel responses to Cucurbitacin I hydrate, D-(+)-sucrose octaacetate and Azadirachtin; I-a sensilla also display significantly increased responses to (-)-lobeline HCl, Denatonium benzoate and Berberine chloride; I-b sensilla also display significantly decreased responses to Theobromine, Caffeine, Theophylline and Umbelliferone; S-a sensilla display significantly increased responses to Cucurbitacin I hydrate and D-(+)-sucrose octaacetate, and significantly decreased responses to Denatonium benzoate, Sparteine sulfate salt, Strychnine nitrate salt, Aristolochic acid, Berberine chloride, and (-)-lobeline HCl; these labellar sensilla types display no significant electrophysiological response changes to Rotenone, N,N-Diethyl-m-toluamide, Escin, Gossypol, Myricetin, Coumarin, Saponin, and Quinine.

Scer\GAL4^Gr66a.1.8/Gr22b^UAS.cDa is an enhancer of abnormal neurophysiology phenotype of Gr59c^Δ

Gr22b^UAS.cDa, Gr59c^Δ, Scer\GAL4^Gr66a.1.8 has abnormal neurophysiology phenotype

Scer\GAL4^Gr66a.1.8/Gr22b^UAS.cDa is an enhancer of intermediate labellar taste bristle phenotype of Gr59c^Δ

Gr22b^UAS.cDa, Gr59c^Δ, Scer\GAL4^Gr66a.1.8 has intermediate labellar taste bristle phenotype