A recessive background phenotype affecting glomerular organization of Or47b olfactory receptor neurons is associated with chromosomes carrying the P{CaryP}Msp300attP40 insertion or derivatives of this docking site. The phenotype is independent of Msp300 function and is likely caused by an effect on other nearby genes affected by the insertion at the attP40 site, and/or a second-site linked mutation. In addition, the attP40 background appears to interact with chromosomes carrying the P{Or47b-GAL4.7.467}Bacc15.5A insertion, producing a similar, but qualitatively distinguishable glomerular defect from the one seen in homozygotes for the attP40 background. The genetic mechanisms underlying this interaction are not known.
The P{CaryP}Msp300attP40 docking site is located within an intron or upstream of Msp300, depending on the isoform, and may disrupt the function of the gene in homozygotes.
A recessive background phenotype affecting glomerular organization of Or47b olfactory receptor neurons is associated with chromosomes carrying the P{CaryP}Msp300attP40 insertion or derivatives of this docking site. The phenotype is independent of Msp300 function and is likely caused by an effect on other nearby genes affected by the insertion at the attP40 site, and/or a second-site linked mutation. In addition, the attP40 background appears to interact with chromosomes carrying the P{Or47b-GAL4.7.467}Bacc15.5A insertion, producing a similar, but qualitatively distinguishable glomerular defect from the one seen in homozygotes for the attP40 background. The genetic mechanisms underlying this interaction are not known.
The P{CaryP}Msp300attP40 docking site is located within an intron or upstream of Msp300, depending on the isoform, and may disrupt the function of the gene in homozygotes.
phiC31\attP docking site for phiC31\int mediated integration into the genome.
Intergenic: flanked by CG14035 and Msp-300.