FB2026_02 , released June 18, 2026
Aberration: Dmel\Df(3R)T-47
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General Information
Symbol
Df(3R)T-47
Species
D. melanogaster
Name
FlyBase ID
FBab0002658
Feature type
chromosomal_deletion
Computed Breakpoints include

86F1-86F2;87A9

Features within 86F1--87A9
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Tp(1;3)TE86Ea
Mutagen
Class of aberration (relative to wild type)
chromosomal_deletion
(FlyBase, 2007)
Class of aberration (relative to progenitor)
chromosomal_deletion
(Hu et al., 1998, Glover et al., 1995)
Breakpoints

86E12-86E20;87A9

(Glover et al., 1995)

86F1-86F2;87A9

(Hu et al., 1998)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

mgr << bk1 << l(3)86Fd << bk2 hits l(3)87Ae

Genetic mapping information
Comments
Comments on Cytology

Ref: Gausz et al., 1981, Genetics 98(4): 775--789.

(Glover et al., 1995)

Limits of break 1 from polytene analysis (FBrf0101924) Left limit of break 2 from inclusion of l(3)87Ae (FBrf0102100) Right limit of break 2 from polytene analysis (FBrf0080055)

Sequence Crossreferences
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations

    Heterozygotes with Df(3R)kar-D1 exhibit normal development of the CNS midline cells, VUM neuron axons and tracheal system. The CNS axon scaffold is mutant in some embryos, embryos show a disorganisation of axons with poorly separated commissures. In combination with Abl1 mutants exhibit severe axonal defects in which the longitudinals and commissures are thin or absent. Axons are disorganised and bulging.

    (Hu et al., 1998)
    NOT in combination with other aberrations
    Stocks (0)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     
    Synonyms and Secondary IDs (3)
    Reported As
    Symbol Synonym
    Df(3R)T-47
    Df(3R)T47
    (Giet et al., 2002)
    Df(3R)TE47
    Name Synonyms
    Secondary FlyBase IDs
      References (9)