Imprecise excision of P{SUPor-P}Atg8aKG07569 removes the first 25 codons of Atg8a and the first 53 codons of BTBD9.
Df(1)Atg8ad4 mutants are homozygous viable and fertile with no gross morphological or developmental abnormalities.
Df(1)Atg8ad4 mutants display basal autophagy defects in larval fat body cells.