P-element-induced male recombination, resulting in an 18kb deletion proximal to the insertion site.
Homozygotes are viable and fertile.
Df(3L)atk33 mutants show defective morphology of embryonic lch5 organs: cilia are not parallel to each other but point out in different directions (though they still contact the cap cell); neuronal cell bodies appear frequently disorganized.
Df(3L)atk33 mutants also show defective morphology of larval lch5 organs: cap and ligament cells are normally stretched but neuronal cilia are frequently disorganized.
Mutant larvae exhibit severe defects in locomotion: the number of direction changes is significantly increased, and the total path length is significantly reduced.
Df(3L)atk33 mutant larvae show a reduced response to sucrose in a chemotaxis assay.
