Somatic clones (induced specifically in the eye) of Vav^G0147 homozygous cells in pupal retina display supernumerary cells in ommatidia (including cone cells and primary pigment cells).

Defects in axon projection are seen in approximately 14% of stage 17 vav^G0147 embryos. On average two longitudinal axons are misrouted across the midline in each embryo.

All vav^G0147 homozygous mutant embryos show disruption to photoreceptor targeting. There is an uneven spread of axon connections in the lamina; some regions are hyperinnervated whilst others lack innervation. Axonal defects are also seen in the medulla, ranging from a total lack of medullar innervation to an increase in the number of neurons entering the medulla region compared to wild type.