Carboxy-terminal truncation of the RhoGEF64C sequences.
Expression of Gef64CΔC.Scer\UAS under the control of Scer\GAL4elav.PLu does not result in a gain of function defective axon guidance phenotype.
RhoGEF64CΔC.UAS, Scer\GAL4ems.HRE is a non-suppressor of posterior spiracle primordium phenotype of Rho1N19.UAS, Scer\GAL4ems.HRE
Co-expression of Gef64CΔC.Scer\UAS does not rescue the invagination defects seen in the posterior spiracles of embryos expressing Rho1N19.Scer\UAS under the control of Scer\GAL4ems.HRE.