Pan-neuronal expression of the diffusible tutlGH15753.Scer\UAS isoform under the control of Scer\GAL4elav-C155 produces Fas2-positive tracks that crisscross the midline. The same phenotype is produced upon expression in the midline, driven by Scer\GAL4sim.PS.
Overexpression of tutlAT02763.Scer\UAS under the control of Scer\GAL4sca-109-68, Scer\GAL4how-24B or Scer\GAL4G14 produces excessive branching of the ISNd nerve, stalling of motor nerves, with some nerves, such as the transverse nerve, innervating muscles that are not their normal targets.
Overexpression of tutlAT02763.Scer\UAS in a wild-type background using the retina-specific Scer\GAL4GMR.PF produces neuronal defects in that many retinal axons stall before reaching their targets and some sprout extra axonal processes that can invade the cortex.
Pan-neuronal overexpression of tutlAT02763.Scer\UAS under the control of Scer\GAL4elav-C155 promotes neuronal invasiveness, with some neuronal cell bodies invading the normally cell-free neuropil layer.