Female cdc14^Δ1 homozygotes, crossed to male cdc14^Δ1 homozygotes, do not have defects in egg laying; cdc14^Δ1 homozygous progeny from this cross do not have defects in survival rate or sex ratio compared to controls. Male cdc14^Δ1 homozygotes produce a similar number of progeny to control males when crossed to control females; they also have no apparent defects in spermatogenesis or sperm morphology. A similar proportion of cdc14^Δ1 mate at least once within 24 hours compared to controls.

cdc14^Δ1 homozygous embryos do not have alterations in aspect ratio. Following UV radiation exposure, cdc14^Δ1/cdc14^Δ1 larvae have similar levels of DNA damage (TUNEL staining) in the wing disc and similar reductions in mitosis (PH3 staining) in the eye disc, compared to controls.

cdc14^Δ1/cdc14^Δ1 does not cause larval locomotion defects or adult climbing defects relative to controls; cdc14^Δ1/cdc14^Δ1 larvae also have no difference in light sensitivity compared to controls.

cdc14^Δ1 homozygous larvae show a significantly reduced touch response, with a broader distribution, compared to controls; they also feed significantly less and have significantly impaired bitter (quinine) avoidance, but no alteration in sugar-bitter preference, with no sex differences; touch response and bitter avoidance are partially rescued by maternally contributed cdc14 protein. In contrast, cdc14^Δ1 homozygous adults have no difference in overall feeding rate or quinine avoidance compared to controls.

Fat bodies from cdc14^Δ1 homozygous larvae have significantly larger, abnormally shaped, lipid droplets, compared to controls.

cdc14^Δ1 homozygous adult females have significantly shorter survival under starvation conditions than controls.

Males homozygous for cdc14^Δ1 have a significant reduction, compared to controls, in the proportion of their progeny, relative to the progeny of an additional (control) male mated to the same female; this effect occurs irrespective of mating order.