The following information accompanied a stock donated to the Bloomington Stock Center by Adelaide Carpenter, Cambridge University (4/02).
In(3L)A54 was induced by X-ray treatment of a st1 pp red1 e4 chromosome. Its breakpoints are 65A7-15;69B3-C11;72E3-F4 and its new cytological order is:
3Lt \- 65A7 | insertion of 6 bands of unknown origin | 69B3 \- 65A15 | 72E3 \- 69C11 | 72F4 \-3Rt.
The inversion complex is associated with homozygous lethality and a dominant gapped wing vein phenotype involving L4.
In combination with Df(3L)E44 or Df(3L)F10, recessive phenotypes are seen. These include missing mid-thorax, reduced eyes and aristae, and held-up wings. These recessive phenotypes may reflect effects of the 69BC inversion breakpoint on gv and eyg. Viability is low with many uneclosed pupae. When dissected, these pupae are headless (except for the presence of palps) or have very reduced heads. They also show the thoracic defects seen in the viable escapers.
The X-ray treatment also induced a new allele of cu, called cu2, identified by its curled wing phenotype in combination with cu1. There is no cytologically visible aberration in region 86 where the cu locus maps. cu2 can be separated from the inversion complex by meiotic recombination.