The arista of th1 flies lacks all lateral branches.
Homozygous mutant or th1/Df(3L)th102 animals have abnormal aristae, they show an almost complete lack of branching, though the strength of this phenotype is variable. Often, a few short extensions (usually shorter than the dorsal laterals) can be seen projecting from various locations along the central core, though not in any clear pattern. In the antennal imaginal discs of third instar larvae, there is a considerable increase in the number of apoptotic cells near the aristal progenitor cell cluster compared to wild-type. But at other stages, cell death appears normal.
Arista lack the normally occurring side branches.
Geotaxis score significantly lower than for Canton S in a Hirsch-type geotaxis maze.
Aristae threadlike, without side branches. Mutation affects development of tarsal claws at 29oC (Mglinetz and Ivanov, 1975). Deficiency for th homozygous lethal (Ward and Alexander, 1957; Korge, 1972). Mutant males and females less successful in mating than wild-type (Burnet, Connolly and Dennis, 1971).
Diap1th-1/th[+] is an enhancer of visible phenotype of LrrkI1915T.UAS, Scer\GAL4GMR.PF, foxoUAS.cKb
Diap1th-1/th[+] is an enhancer of eye phenotype of LrrkI1915T.UAS, Scer\GAL4GMR.PF, foxoUAS.cKb
The eye phenotype caused by co-expression of foxoScer\UAS.cKb and LrrkI1915T.Scer\UAS under the control of Scer\GAL4GMR.PF is enhanced by th1/+.
Diap1th-1 is partially rescued by Scer\GAL4T80/Diap1UAS.cHa
Bridges, 31st Oct. 1922.