Homozygous cells in the morphogenetic furrow (in clones that encompass the morphogenetic furrow) show a dramatic loss of apical constriction and apicobasal contraction.

When homozygous mutant clones are made in the eye, clones away from the equator are generally well ordered, with only occasional defects such as ommatidial rotation errors of less than 45^o. Increased disorder is seen if clones are near the equator, where missing photoreceptors are often observed, and the array is often less regular. 70.8% of ommatidia in homozygous mutant somatic clones in the eye are normal. 17.1% have rotated ommatidia, 0.2% have chirality defects, 0.3% are achiral (11.6% unscorable). When Rho1^72M1 clones are made in the eye that create mosaic ommatidia, chirality defects are only seen (7.4% of cases) when presumptive R3 cells are mutant and presumptive R4 cells are wild-type.

Homozygous clones in the eye produce ommatidia that are incorrectly rotated relative to the equator and sometimes have inappropriate chiral forms. Loss of photoreceptors is also occasionally seen.