Large Df(2L)32FP-5 mutant clones (which cause the salr^32FP-5 allele and removes salm and apparently no other gene), in the adult male genitalia cause the loss of the posterior lobe, lateral plate and genital arch. No other external genitalia structures are affected. Males with clones also exhibit defects in the testis. Testes are smaller, lighter in colour and abnormally coiled.

No dorsal trunk forms in the tracheal system in Df(2L)32FP-5 (removes salm and causes salr^32FP-5) embryos.

Large mitotic clones of cells homozygous for Df(2L)32FP-5 (which uncovers both salm and salr) result in reorganisation of the venation pattern that are manifest both within and outside the salm/salr domain of expression. Small clones localized between the veins L2 and L3, and between L4 and L5 cause autonomous formation of ectopic vein tissue, irrespective of the wing surface where the clones appear. The differentiation of L3 and L4 is not affected in these clones, rather these veins are displaced. Clones in the anterior compartment can also differentiate ectopic sensilla characteristic of L3. All clones that span wing vein L2 result in the elimination of this vein.

100% of flies carrying Df(2L)32FP-5 and either Df(2L)FCK-20, T(2;3;4)FCK-25 or In(2L)FCK-73 lack the anterior notopleural macrochaetae and humeral chaetae, and ectopic chaetae are seen on the base of the first leg in 2.9%, 3.7% and 3.5% of flies respectively.