FlyBase Allele Report: Dmel\Hr39[c739]

General Information

Symbol

Dmel\Hr39^c739

Species

D. melanogaster

Name

FlyBase ID

FBal0243538

Feature type

allele

Associated gene

Dmel\Hr39

Associated Insertion(s)

P{GawB}Hr39^c739

Carried in Construct

Also Known As

c739-GAL4, c739, c739 GAL4, GAL4-c739, c739GAL4

Key Links

Allele class

Mutagen

P-element activity

Nature of the Allele

Allele class

Mutagen

P-element activity

(Bates et al., 2010)

Progenitor genotype

Associated Insertion(s)

P{GawB}Hr39^c739

Cytology

Description

P{GawB}Hr39^c739 is located in the second intron of Hr39, approximately 9.5kb upstream of the ATG start codon.

(Boulanger et al., 2011)

P{GawB}Hr39^c739 is inserted in the second intron of Hr39.

(Bates et al., 2010)

Allele components

Component

Use(s)

Inserted element

P{GawB}

enhancer trap

Encoded product / tool

GAL4

binary expression system - driver

Mutations Mapped to the Genome

Curation Data

Type

Location

Additional Notes

References

Variant Molecular Consequences

Associated Sequence Data

DDBJ / EMBL / GenBank

DNA sequence

Protein sequence

UniProtKB/Swiss-Prot

UniProtKB/TrEMBL

Expression Data

Reporter Expression

Additional Information

Statement

Reference

Marker for

Reflects expression of

Reporter construct used in assay

Human Disease Associations

Disease Ontology (DO) Annotations

Models Based on Experimental Evidence ( 0 )

Disease

Evidence

References

Modifiers Based on Experimental Evidence ( 0 )

Disease

Interaction

References

Comments on Models/Modifiers Based on Experimental Evidence ( 0 )

Disease-implicated variant(s)

Phenotypic Data

Phenotypic Class

Phenotype Manifest In

Detailed Description

Statement

Reference

External Data

Linkouts

Interactions

Show genetic interaction network for Enhancers & Suppressors

Phenotypic Class

NOT Enhanced by

Statement

Reference

Hr39^c739, babo^unspecified has abnormal neuroanatomy phenotype, non-enhanceable by ftz-f1^β.UAS.cBa/Scer\GAL4^Tab2-201Y

(Boulanger et al., 2011)

Suppressed by

Statement

Reference

Hr39^c739, babo^unspecified has abnormal neuroanatomy phenotype, suppressible | partially by Scer\GAL4^Tab2-201Y/EcR^B1.UAS

(Boulanger et al., 2011)

NOT suppressed by

Statement

Reference

Hr39^c739, babo^unspecified has abnormal neuroanatomy phenotype, non-suppressible by ftz-f1^β.UAS.cBa/Scer\GAL4^Tab2-201Y

(Boulanger et al., 2011)

NOT Enhancer of

Statement

Reference

Hr39^c739 is a non-enhancer of abnormal neuroanatomy phenotype of babo^unspecified

(Boulanger et al., 2011)

NOT Suppressor of

Statement

Reference

Hr39^c739 is a non-suppressor of abnormal neuroanatomy phenotype of babo^unspecified

(Boulanger et al., 2011)

Phenotype Manifest In

NOT Enhanced by

Statement

Reference

Hr39^c739, babo^unspecified has gamma Kenyon cell phenotype, non-enhanceable by ftz-f1^β.UAS.cBa/Scer\GAL4^Tab2-201Y

(Boulanger et al., 2011)

Suppressed by

Statement

Reference

Hr39^c739, babo^unspecified has gamma Kenyon cell phenotype, suppressible | partially by Scer\GAL4^Tab2-201Y/EcR^B1.UAS

(Boulanger et al., 2011)

NOT suppressed by

Statement

Reference

Hr39^c739, babo^unspecified has gamma Kenyon cell phenotype, non-suppressible by ftz-f1^β.UAS.cBa/Scer\GAL4^Tab2-201Y

(Boulanger et al., 2011)

NOT Enhancer of

Statement

Reference

Hr39^c739 is a non-enhancer of gamma Kenyon cell phenotype of babo^unspecified

(Boulanger et al., 2011)

NOT Suppressor of

Statement

Reference

Hr39^c739 is a non-suppressor of gamma Kenyon cell phenotype of babo^unspecified

(Boulanger et al., 2011)

Additional Comments

Genetic Interactions

Statement

Reference

Xenogenetic Interactions

Statement

Reference

Complementation and Rescue Data

Comments

Images (0)

Mutant

Wild-type

Stocks (2)

Bloomington

7362

y¹ w^67c23; P{GawB}Hr39^c739

64305

y¹ w^67c23; P{GawB}Hr39^c739 P{UAS-mCD8::GFP.L}LL5

Notes on Origin

Discoverer

External Crossreferences and Linkouts ( 0 )

Synonyms and Secondary IDs (1)

Reported As

Symbol Synonym

Hr39^c739

Name Synonyms

Secondary FlyBase IDs

References (2)

Report Sections

Open Close