FB2026_01 , released March 12, 2026
FB2026_01 , released March 12, 2026
Allele: Dmel\commKR.UAS.Tag:HA
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General Information
Symbol
Dmel\commKR.UAS.Tag:HA
Species
D. melanogaster
Name
FlyBase ID
FBal0264530
Feature type
allele
Associated gene
Dmel\comm
Associated Insertion(s)
Carried in Construct
P{UAS-comm.KR.HA}
Key Links
Transgenic product class
inframe_deletion
(Keleman et al., 2005)
missense_variant
(Keleman et al., 2005)
Mutagen
Nature of the Allele
Transgenic product class
inframe_deletion
(Keleman et al., 2005)
missense_variant
(Keleman et al., 2005)
Mutagen
in vitro construct
(Keleman et al., 2005)
Progenitor genotype
commΔ245-370.UAS.Tag:MYC
(Keleman et al., 2005)
Carried in construct
P{UAS-comm.KR.HA}
Cytology
Description

The six remaining comm lysine residues in commΔ245-370.UAS.Tag:MYC have been mutated to arginine and the Tag:MYC tag has been replaced with Tag:HA.

(Keleman et al., 2005)
Allele components
Component
Use(s)
Regulatory region(s)
UASt
Encoded product / tool
comm
Tagged with
Tag:HA
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
 
UniProtKB/Swiss-Prot
    UniProtKB/TrEMBL
      Expression Data
      Reporter Expression
      Additional Information
      Statement
      Reference
       
      Marker for
      Reflects expression of
      Reporter construct used in assay
      Human Disease Associations
      Disease Ontology (DO) Annotations
      Models Based on Experimental Evidence ( 0 )
      Disease
      Evidence
      References
      Modifiers Based on Experimental Evidence ( 0 )
      Disease
      Interaction
      References
      Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
       
      Disease-implicated variant(s)
       
      Phenotypic Data
      Phenotypic Class
      Phenotype Manifest In
      Detailed Description
      Statement
      Reference

      Expression of commKR.Scer\UAS.T:Ivir\HA1 under the control of Scer\GAL4ap-md544 causes the ipsilateral ap neurons to ectopically cross the midline.

      (Keleman et al., 2005)
      External Data
      Interactions
      Show genetic interaction network for Enhancers & Suppressors
      Phenotypic Class
      Phenotype Manifest In
      Additional Comments
      Genetic Interactions
      Statement
      Reference
      Xenogenetic Interactions
      Statement
      Reference
      Complementation and Rescue Data
      Rescues

      commKR.UAS.Tag:HA/Scer\GAL4Poxn.14 rescues comm072

      (Keleman et al., 2005)
      Comments

      Expression of commKR.Scer\UAS.T:Ivir\HA1 in the poxn neurons under the control of Scer\GAL4Poxn.14 rescues the midline crossing defects seen in comm072 homozygous stage 16 embryos.

      (Keleman et al., 2005)
      Images (0)
      Mutant
      Wild-type
      Stocks (0)
      Notes on Origin
      Discoverer
      External Crossreferences and Linkouts ( 0 )
      Synonyms and Secondary IDs (2)
      Reported As
      Symbol Synonym
      commKR.Scer\UAS.T:Ivir\HA1
      commKR.UAS.Tag:HA
      Name Synonyms
      Secondary FlyBase IDs
        References (1)