Pol32^L2, DNApol-ζ^3B double mutants show more pronounced DNA repair defects in a P{w[α]} assay than Pol32^L2 single mutants: more severely decreased homologous recombination events than Pol32^L2 and increased incomplete homologous recombination events that terminate via end joining, as compared to wild-type controls.

Pif1¹⁶⁷, DNApol-ζ^3B individuals do not show significant DNA repair defects in P{w[α]} assays: slight decrease in homologous recombination events and unchanged incomplete homologous recombination events that terminate via end joining, as compared to controls.

DNApol-η¹² mus205^3B double mutants show no difference in the frequency of full homologous recombination (HR) repair events in assays of excision and repair events of P{hsw^a} compared to wild type. The repair tract lengths in aborted HR products isolated from the double mutants are substantially increased compared to wild type.

Pol32^L2 mus205^3B double mutants show an approximately 70% decrease in full homologous recombination (HR) repair events in assays of excision and repair events of P{hsw^a} (similar to the reduction seen in Pol32^L2 single mutants). Repair synthesis tract lengths are reduced dramatically in aborted HR repair products compared to wild type, and this defect is more severe than that seen in Pol32^L2 single mutants.