The T2A-GAL4 sequence present in TI{2A-GAL4}Dop1R1PGF3 has been replaced with a T2A-lexA::UnkAD sequence, resulting in the lexA::UnkAD driver being expressed as a separate protein under the control of the endogenous Dop1R1 sequences. An attL site is present upstream of the lexA::UnkAD sequence, and a loxP site and an FRT3 site are present downstream of the lexA::UnkAD sequence.