12E9-12E11;13A9-13B1
12E9-12E11;13A9-13B1
12E9-12E11;13B2-13B5
mud << bk1 << NetA << eag << bk2
Embryos exhibit variable defects in the formation of commissural pathways. Scer\GAL4rho.PL induced expression of P{UAS-NETA} or P{UAS-NETB} significantly rescued the CNS phenotype.
CNS defect in embryos: axon commissures are drastically thinner or missing amd the longitudinal tracts are highly disorganised and discontinuous.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
W.R. Engels.
1B;12A6-12A10;13A2-13A5;20
All limits from polytene analysis (FBrf0080317)