[17D1-17D1];[17D3-17D3];;[17D1-17D1];[17D3-17D3];
17D1;17D3
17D1;17D3;17D1;17D3
Breakpoint from FlyBase's release 5 sequence location of progenitor insertion.
Dp(1;3)DC351 completely rescues the Df(1)Exel9051 mutant neuromuscular junction (NMJ) phenotype.
Dp(1;3)DC352 fails to rescue the Df(1)Exel9051 mutant neuromuscular junction (NMJ) phenotype.
Compared with wild-type, Df(1)Exel9051/Df(1)ED7413 mutant larvae exhibit neuromuscular junction (NMJ) undergrowth.
Inferred to overlap with: Df(1)ED7413.
The size and frequency of miniature excitatory junction potentials (mEJPs) from muscle 6 of Df(1)Exel9051/Y mutants does not differ from that of wild-type. However, the size of excitatory junction potentials (EJPs) is reduced in Df(1)Exel9051/Y mutants compared with wild-type.
Df(1)Exel9051 is viable when hemi- or homozygous. Compared with wild-type, homo- or hemizygous Df(1)Exel9051 mutant larvae exhibit neuromuscular junction (NMJ) undergrowth.
Under dim light conditions, the distance travelled by mutant third instar larvae in one minute at 20-22[o]C is not significantly different from wild type. The frequency of peristaltic contractions under these conditions is normal in the mutant larvae. However, under bright light at 27-30[o]C (stress conditions) the mutant larvae travel significantly less far than wild type in one minute. Under these conditions, the frequency of peristaltic contractions is reduced in the mutant larvae compared to wild type.
The olfactory receptor neurons of Df(1)Exel9051 mutant flies do not show any overt phenotype, either in uninjured flies or in flies in which the antennae have been surgically ablated.
FlyBase curator comment: The aberration chromosome previously referred to as "Df(1)Exel9054" is in fact identical to Df(1)Exel9051 (generated using the same progenitor insertions), and thus the two entries have been merged in the database.
Limits computationally determined from location of progenitor P insertion on genome sequence between P{EP}EP443EP443/P{EP}BxEP1383 and P{EP}Pk17EEP438