15% of stage 11-13 vri²/vri^k05901 embryos show tracheal defects. During stages 14-17, 80% of vri²/vri^k05901 embryos show weak tracheal defects (the general architecture of the trachea is preserved, but breaking of branches occurs and elongation and branching are abnormal) and 12% show strong tracheal defects (the tracheal architecture is strongly affected with complete disorganization and formation of sacs).

54% of stage 11-13 vri²/vri^5R7.2 embryos show tracheal defects. During stages 14-17, 76% of vri²/vri^5R7.2 embryos show weak tracheal defects (the general architecture of the trachea is preserved, but breaking of branches occurs and elongation and branching are abnormal) and 12% show strong tracheal defects (the tracheal architecture is strongly affected with complete disorganization and formation of sacs).

Homozygous border cell clones show only subtle delays in migration.

Bristles in homozygous clones are thinner and reduced in size than normal or are missing. The average distance between stout bristles of the triple row is reduced compared to wild type in homozygous clones, which is characteristic of smaller cells.

Homozygous embryos are shortened and the dorsal epidermis often appears wrinkled and reduced (leading to a slight 'tail-up' phenotype) and trachea are interrupted. Less frequently the head skeleton is abnormal and ventral denticles are fused or missing. Hemizygotes die as larvae. vri²/vri³ transheterozygotes exhibit shortening of wing vein L5.

vri²/vri[+] is an enhancer of wing phenotype of dpp^hr4/dpp^d6

vri² is an enhancer of phenotype of ea^161.13

Mad⁶, vri² has wing vein L5 phenotype

Mad⁶, vri² has wing vein | ectopic phenotype

dpp^hr4/dpp^d6, vri²/vri[+] has leg phenotype

dpp^hr4/dpp^d6, vri²/vri[+] has wing vein phenotype

Mad⁶, vri² has posterior crossvein phenotype

dpp^hr4/dpp^d6, vri² has eye phenotype

dpp^hr4/dpp^d6, vri² has leg phenotype

dpp^hr4/dpp^d6, vri² has wing vein phenotype

dpp^hr4/dpp^d6, vri²/vri[+] has eye phenotype