Pcl¹/Pcl⁴ males show transformation of the second leg to first leg (sex comb teeth are found on the second leg).

4% of heterozygotes show transformation of abdominal segment A4 to A5 (extra black pigmentation is seen on the fourth abdominal tergite in males). 0% of heterozygotes show transformation of abdominal segment A6 to A7 (males have a reduced sixth abdominal tergite). There is an average of 0.2 extra sex combs (average total number of extra sex comb teeth on the four posterior legs of a male).

Shows no dominant effect on telomeric Position Effect Variegation (PEV) in stocks carrying a variegating w^+mW.hs allele at the telomeres of the second and third chromosomes.

Embryonic lethal or semi-lethal when heterozygous with ph-d². Unhatched larvae carry segmentation defects or polarity defects.

Homozygous embryos show posteriorly directed transformations; A1 resembles A2 and A2-7 transformed into more posterior segments, head and thorax normal. Pcl¹ Scm¹ double homozygotes exhibit transformation of all segments toward A8; Keilin's organs retained in thoracic segments; head involution does not occur; prominent abdominal denticle band seen on dorsal surface of head. Homozygous embryos derived from transplanted homozygous maternal germ-cell precursors display A8 morphology in all thoracic and abdominal segments; head seems to resemble that of Pcl¹ Scm¹ double homozygotes produced by heterozygous mothers; heterozygous adults from homozygous oocytes show patchy transformations of A4 to A5 and of A6 and A7 to A8; similar transformation infrequently seen in heterozygous offspring of heterozygous mothers.

Heterozygotes often show weak transformation of the second and third legs to first legs, and partial transformation of the fourth abdominal segment to fifth abdominal segment. Hemizygotes and homozygotes die as embryos that appear to complete embryogenesis and have normal cuticle structures. Homozygous clones appear to have normal viability in the abdominal tergites. These clones are often transformed to more posterior segments in the second to sixth abdominal segments. Homozygous clones in the first abdominal segment do not show transformations. Homozygous clones in the wing have normal viability and often appear to be partially transformed to haltere, developing small, densely packed trichomes and occasionally small bristles characteristic of the haltere. Pcl¹/Pcl³ flies usually die as pharate adults. Head structures usually appear normal, although, rarely, outgrowths of leg tissue (as seen by the presence of bracted bristles) occur on the antennae. The prothorax, including the first leg, and humerus, mesonotum and dorsal metathorax appear normal. Wing vein L5 often fails to reach the wing margin, possibly indicating a weak transformation of wing to haltere. Almost the entire anterior compartment of the second leg is transformed into a first leg anterior compartment in most cases. The posterior compartment of the second leg contains a mixture of first, second and third leg posterior structures. Transformation of the third leg to first leg occurs mainly in the anterior compartment. This transformation is complete distally, with sex combs of 9-13 teeth forming in the basitarsus, but is incomplete proximally, with the coxa being only slightly affected. The posterior compartment of the third leg appears almost normal, although there is some indication of transformation to first or second leg; occasionally a bristle forms in the central region of the trochanter posterior compartment, small groups of trichomes often occur in the central portion of the femur posterior compartment, and the bristles at the ventral edge of this region are slightly enlarged. All the abdominal tergites of Pcl¹/Pcl³ flies show variegated transformation towards more posterior segments. The 1st abdominal tergite often contains patches of cuticle that are transformed to 2nd, 3rd or perhaps 4th tergite. The 2nd, 3rd and 4th tergites often show patches of cuticle that are transformed to 5th or 6th tergite. The 5th tergite is usually transformed to 6th tergite, and the 6th tergite is usually transformed to 7th tergite, which results in the loss of the 6th tergite in males. The external genitalia and analia are normal in Pcl¹/Pcl³ flies. Internally, females show transformation of the parovaria into spermathecae. The internal genitalia of males are normal.

Strong interaction with Pc³. homozygous lethal; enhances Pc³

Pcl⁴/Pcl¹ has visible | homeotic phenotype, suppressible by Gug[+]/Gug¹⁷⁵⁷

Pcl⁴/Pcl¹ has visible | homeotic phenotype, suppressible by Gug³³³³/Gug[+]

Pcl⁴/Pcl¹ has visible | homeotic phenotype, suppressible by Gug⁵¹⁹/Gug[+]

Hsc70-4^54.1, Pcl¹ has visible | dominant | homeotic phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has visible | dominant | homeotic phenotype

Pcl¹ has sex comb | ectopic phenotype, enhanceable by Hsc70-4[+]/Hsc70-4^54.1

Pcl¹ has phenotype, enhanceable by Dp(3;3)D1

Pcl¹ has phenotype, enhanceable by Df(3R)P9

Pcl¹ has phenotype, enhanceable by Dp(3;3)P5

Pcl⁴/Pcl¹ has sex comb | ectopic phenotype, suppressible by Gug³³³³/Gug[+]

Pcl⁴/Pcl¹ has sex comb | ectopic phenotype, suppressible by Gug⁵¹⁹/Gug[+]

Pcl⁴/Pcl¹ has mesothoracic leg phenotype, suppressible by Gug[+]/Gug¹⁷⁵⁷

Pcl⁴/Pcl¹ has mesothoracic leg phenotype, suppressible by Gug³³³³/Gug[+]

Pcl⁴/Pcl¹ has mesothoracic leg phenotype, suppressible by Gug⁵¹⁹/Gug[+]

Pcl⁴/Pcl¹ has prothoracic leg | ectopic phenotype, suppressible by Gug[+]/Gug¹⁷⁵⁷

Pcl⁴/Pcl¹ has prothoracic leg | ectopic phenotype, suppressible by Gug³³³³/Gug[+]

Pcl⁴/Pcl¹ has prothoracic leg | ectopic phenotype, suppressible by Gug⁵¹⁹/Gug[+]

Pcl⁴/Pcl¹ has sex comb | ectopic phenotype, suppressible by Gug[+]/Gug¹⁷⁵⁷

Pcl¹ has phenotype, suppressible by Dp(3;3)P5

Pcl¹ has phenotype, suppressible by Dp(3;3)C126

Pcl¹ is an enhancer of wing phenotype of Ubx^Cbx-1

Pcl¹ is an enhancer of phenotype of Antp^Scx

Df(2R)Pcl11B/Pcl¹ is an enhancer of embryonic segment phenotype of Pc³

Df(2R)Pcl11B/Pcl¹ is an enhancer of mouth hook phenotype of Pc³

Df(2R)Pcl11B/Pcl¹ is an enhancer of embryonic head phenotype of Pc³

Pcl¹ is an enhancer of phenotype of Abd-B^Mcp-1

Pcl¹ is an enhancer of phenotype of Scr^Msc

Hsc70-4^54.1, Pcl¹ has adult abdominal segment 4 phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has adult abdominal segment 4 phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has adult abdominal segment 6 phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has adult abdominal segment 5 | ectopic phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has abdominal tergite 6 | male phenotype

Hsc70-4^54.1, Pcl¹ has adult abdominal segment 6 phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has abdominal tergite 4 | male phenotype

Hsc70-4[+]/Hsc70-4^54.1, Pcl¹ has adult abdominal segment 7 | ectopic phenotype

Hsc70-4^54.1, Pcl¹ has adult abdominal segment 5 | ectopic phenotype

Hsc70-4^54.1, Pcl¹ has abdominal tergite 6 | male phenotype

Hsc70-4^54.1, Pcl¹ has abdominal tergite 4 | male phenotype

Hsc70-4^54.1, Pcl¹ has adult abdominal segment 7 | ectopic phenotype

Pcl¹, Scm¹ has larval abdominal segment 3 phenotype

Pcl¹, Scm¹ has larval abdominal segment 5 phenotype

Pcl¹, Scm¹ has larval abdominal segment 6 phenotype

Pcl¹, Scm¹ has larval abdominal segment 8 | ectopic phenotype

Pcl¹, Scm¹ has larval abdominal segment 1 phenotype

Pcl¹, Scm¹ has larval abdominal segment 2 phenotype

Pcl¹, Scm¹ has larval abdominal segment 4 phenotype

Pcl¹, Scm¹ has larval abdominal segment 7 phenotype