When rpr^Scer\UAS.cJa is driven by Scer\GAL4^eve.CQ2 and Scer\GAL4^eve.RN2 the aCC U/CQ RP2 and pCC neurons are ablated. When this is happens the DO3-DO5 and DT1 dendrites are not seen to expand into the regions vacated by the aCC and U/CQ dendrites.

When expression is driven by Scer\GAL4^455.2, hindgut ectoderm cells are killed and the number of HVM cells is reduced with many of the cells dying. The HVM cells that survive are those attached to remaining hindgut ectoderm.

30% of embryos expressing rpr^Scer\UAS.cJa under the control of Scer\GAL4^s.gcm in which longitudinal glia have been ablated have apoptotic futsch-positive neurons. These multiple apoptoses are non-autonomous consequences of glial ablation (in Df(3L)H99 embryos (which are mutant for rpr) which are expressing rpr^Scer\UAS.cJa under the control of Scer\GAL4^s.gcm, only 23% of embryos have a dying cell).

Expression of rpr^Scer\UAS.cJa under the control of Scer\GAL4^ato.3.6 results in the elimination of neurons in the brain that normally express ato. Many flies do not eclose and escaper flies show a 2-3 day delay in eclosion compared to controls.