Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^elav-C155 results in a mild rough eye phenotype.

P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^Mef2.PR results in significantly reduced muscle width, but not length, in third instar larval body wall muscles, as compared to controls.

Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^GMR.PF results in severe eye abnormalities. Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^en-e16E results in severe truncations of the posterior wing compartment. Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^en-e16E or Scer\GAL4^dpp.blk1 results in increased cell death in the wing disc in the region expressing P1\cre. Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^F4 does not result in morphological abnormalities or increased apoptosis in the third larval instar. Expression of P1\cre^Scer\UAS.cHa under the control of Scer\GAL4^prd.RG1 does not result in differences in the extent of cell death, proliferation and differentiation between P1\cre-expressing and non-expressing regions. Basal expression of P1\cre^Scer\UAS.cHa (in the absence of a Scer\GAL4 driver) results in mosaic inactivation of w^P1\loxP (carried in transgene P{loxP(w^+mC)}) due to recombination at the P1\loxP sites flanking the w sequences.