mirr^e48/mirr¹⁸²⁵ mutants survive at a low frequency to first instar larval stages.

In stage 16 mirr^e48/mirr¹⁸²⁵ mutants the vdaB neuron is lost with 34% frequency, and this is preceded by SOP1a loss. However, all other ventral cluster MD neurons, as well as v'ada, are never lost.

In mirr^e48/mirr¹⁸²⁵ mutant vdaB neuronal clones, the dendritic field coverage is significantly reduced to 84% of wild-type, and branches fail to fully fill the body wall. This is not associated with a change in dendritic self-avoidance. However, it is correlated with a similar reduction of dendritic branching to 86% of wild-type.

mirr^e48/mirr¹⁸²⁵ mutant vdaB neuronal clones fall to send axon terminal branches across the midline. In 19% of clones, both branches fail to cross the midline, and in a further 37% only the posterior branch crosses.

Expression of mirr^VDRC.cUa in the neuroepithelium of mirr¹⁸²⁵ heterozygotes (under the control of Scer\GAL4^{neur-GAL4-A101} results in class IV vdaB loss in 18% of cases in stage 16 embryos. No other neuron loss is seen. In third instar larvae this manipulation leads to a reduction in the frequency at which the larvae initiate a rolling response to 22%, compared to 42-46% in controls.

Scer\GAL4^rn-GAL4-5, egr^UAS.cUa, mirr¹⁸²⁵ has visible | adult stage phenotype, enhanceable by CtBP[+]/CtBP^334Δ4

Scer\GAL4^rn-GAL4-5, egr^UAS.cUa, mirr¹⁸²⁵ has visible | adult stage phenotype

Scer\GAL4^rn-GAL4-5, egr^UAS.cUa, mirr¹⁸²⁵ has wing | ectopic phenotype, enhanceable by CtBP[+]/CtBP^334Δ4

mirr¹⁸²⁵/mirr[+] is an enhancer of wing | ectopic phenotype of CtBP^334Δ4, Scer\GAL4^rn-GAL4-5, egr^UAS.cUa

Scer\GAL4^rn-GAL4-5, egr^UAS.cUa, mirr¹⁸²⁵ has wing | ectopic phenotype