UASt regulatory sequences drive expression of a 500bp inverted repeat.
Cip4NIG.15015R, Scer\GAL4C57 has abnormal neuroanatomy phenotype, suppressible by witA12/wit[+]
Cip4NIG.15015R/Scer\GAL4C57 is a non-enhancer of abnormal neuroanatomy phenotype of witB11/witA12
Cip4NIG.15015R/Scer\GAL4C57 is a non-suppressor of abnormal neuroanatomy phenotype of witB11/witA12
Cip4NIG.15015R, Scer\GAL4C57 has neuromuscular junction phenotype, suppressible by witA12/wit[+]
Cip4NIG.15015R, Scer\GAL4C57 has bouton phenotype, suppressible by witA12/wit[+]
Cip4NIG.15015R/Scer\GAL4C57 is a non-enhancer of synapse phenotype of witB11/witA12
Cip4NIG.15015R/Scer\GAL4C57 is a non-enhancer of neuromuscular junction phenotype of witB11/witA12
Cip4NIG.15015R/Scer\GAL4C57 is a non-suppressor of synapse phenotype of witB11/witA12
Cip4NIG.15015R/Scer\GAL4C57 is a non-suppressor of neuromuscular junction phenotype of witB11/witA12
Expression of Cip4NIG.15015R post-synaptically (under the control of Scer\GAL4C57) in witA12/witB11 larvae does not affect overall bouton number or satellite bouton formation.
A witA12 heterozygous background can completely reverse the increase in total bouton number and satellite bouton formation in larvae expressing Cip4NIG.15015R post-synaptically (under the control of Scer\GAL4C57).