5 day old mutant brains expressing Trap1^Scer\UAS.cBa via Scer\GAL4^Act.PU do not show increased reactive oxygen species. Mitochondrial distribution is not significantly altered.

Young mutant males, but not females, expressing Trap1^Scer\UAS.cBa via Scer\GAL4^Act.PU are significantly more resistant to oxidative stress (paraquat exposure) compared to controls.

Median survival is increased in Scer\GAL4^{αTub84B.Switch.PK} Trap1^Scer\UAS.cBa males exposed to paraquat when expression is induced in 40-day old flies, compared to controls.

In both males and females expressing Trap1^Scer\UAS.cBa via Scer\GAL4^Act.PU, survival after heat shock applied at 19 days of age is significantly increased compared to controls.

The lifespan of males and females expressing Trap1^Scer\UAS.cBa via Scer\GAL4^Act.PU or via Scer\GAL4^{αTub84B.Switch.PK} (maintained on RU486), is not significantly different from controls.

Locomotor performance in males and females expressing Trap1^Scer\UAS.cBa via Scer\GAL4^Act.PU is significantly better than controls at 60 and 70 days of age.

Scer\GAL4^Act.PU-mediated expression of Trap1^Scer\UAS.cBa results in increased female fecundity and males produce significantly more progeny at older ages, compared to controls.

Scer\GAL4^elav.PU-mediated expression of Trap1^Scer\UAS.cBa does not significantly enhance oxidative stress resistance to paraquat, nor does it appreciably change lifespan, but it does improve locomotor performance of both sexes at older age points.

RU486-conditional expression of Trap1^Scer\UAS.cBa via Scer\GAL4^{αTub84B.Switch.PK} increases survival after heat shock applied at 20 days of age in males, but not females.