In the odour-induced jump test, homozygous mutants of mys^olfC-x5 are defective in response to iso-amyl acetate but not benzaldehyde or ethyl acetate, when compared to controls.

Mutant synapses at the larval neuromuscular junction show overgrowth.

Mutants exhibit reduced olfactory responses to isoamyl acetate, whether homozygous or in combination with mys^olfC-x3, mys^olfC-x10 or mys^olfC-x17. Homozygous mutants are wild-type in response to ethyl acetate, but show a reduced response in combination with mys^olfC-x3 and mys^olfC-x10.

Larvae show a reduced response to iso-pentyl acetate. Larvae show abnormal responses to certain acetates. Larvae show no significant response to any volume of hexyl acetate.

Homozygous adults show a reduced attraction to iso-amyl acetate compared to wild-type in a slow chemotaxis assay, although they appear to detect ethyl acetate normally. mys^olfC-x3/mys^olfC-x5 and mys^olfC-x10/mys^olfC-x5 flies are defective in their responses to both ethyl acetate and iso-amyl acetate.

normal response to ethyl acetate; reduced to iso-amyl acetate

mys^olfC-x5, sws^olfE-x26 has abnormal smell perception phenotype

if^B2, mys^olfC-x5 has abnormal smell perception phenotype

if^k27e, mys^olfC-x5 has abnormal smell perception phenotype