fng⁵²/Df(3L)ri-XT1 animals that survive to adulthood when grown at 18^oC exhibit a moderate wing phenotype. fng⁵²/fng⁸⁰ animals that survive to adulthood exhibit a strong wing phenotype. fng⁵²/fng¹³ animals that survive to adulthood exhibit a moderate wing phenotype. fng⁵²/fng^M69 animals that survive to adulthood when grown at 18^oC exhibit a strong wing phenotype. Homozygous fng⁵² animals that survive to adulthood exhibit a moderate wing phenotype. fng⁵²/fng² animals that survive to adulthood exhibit a weak wing phenotype.

Homozygous females have ovaries showing 43% compound follicles, 18% with long stalks 82% of follicles have abnormal epithelium. Transheterozygous fng⁵²/fng^M69 females have ovaries showing 33% compound follicles and 22% of follicles have abnormal epithelium. Where follicles do not include the usual 16 germline cells, those with more than usual germline cells lie next to those with fewer than usual germline cells. Along the length of the entire ovariole the oocyte:nurse cell ratio is normal. The abnormalities of number of germline cells in each egg chamber is caused by defects in the enclosure of germline cysts by somatic follicle cells. The long stalk phenotype is sometimes accompanied by a stalk disorganization phenotype.

fng⁵²/fng¹³ flies have small eyes. Tissue loss in the ocellar region is also seen.

Weak wing vein-splitting or 'delta' phenotype but a normal wing margin in heterozygotes.

Reduction in wing margin formation. fng⁸⁰/fng⁵² mutants are weakly viable and exhibit a severe wing margin loss phenotype.

Loss of tissue from the wing margin.

fng⁵² is an enhancer of wing margin phenotype of Nsf2^EQ.UAS, Scer\GAL4^bbg-C96

fng⁵² is an enhancer of wing phenotype of Mmus\Mfng^UAS.cCa, Scer\GAL4^ptc-559.1

fng⁵² is an enhancer of wing phenotype of Mmus\Rfng^UAS.cCa, Scer\GAL4^ptc-559.1