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General Information
Symbol
Df(1)JA26
Species
D. melanogaster
Name
FlyBase ID
FBab0000463
Feature type
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

Rst(1)JH << bk1 << tsg << l(1)11Da << bk2 << sno

Genetic mapping information
Comments

This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).

Comments on Cytology

Limits of break 1 from polytene analysis (FBrf0034402) Left limit of break 2 from inclusion of rad (FBrf0053305) Right limit of break 2 from non-inclusion of sno (FBrf0068459)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    Flies heterozygous for the deletion do not show a Minute bristle phenotype.

    Df(1)JA26 hemizygous embryos exhibit head and thorax defects similar to tsg loss of function mutants.

    With a very low penetrance, Df(1)JA26 hemizygous embryos show subtle defects in segmentation, including partial fusion of denticle band pairs in the register seen for Ten-m mutants.

    Heterozygous P1 stage pupae have a significantly increased heart rate and an increased strength of heart beat rhythmicity compared to controls.

    Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.

    Initially showed maternal enhancement of dpphr4, but effect not due to disruption of discrete element within the deficiency.

    Deficient embryos show an uninterpretable mutant midgut phenotype.

    Homozygous embryos are very abnormal compared to wild-type.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    No significant change in female lethality when in combination with Sxlf1.

    Stocks (2)
    Notes on Origin
    Discoverer

    Lefevre.

     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     
    Synonyms and Secondary IDs (4)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
      References (34)