phyl1/phyl2 animals die at late embryonic or first larval instar stages.
The number of notal microchaetae and microchaetae on abdominal segments 3 and 4 are reduced in phyl2/phyl4 flies. 58% of bristles on the notum are duplicated, having 2 hairs emerging from 2 sockets or a fused socket, and 9.2% of bristles on the notum show other defects (including 1 hair surrounded by 2-3 sockets, and 2 socket or 4 socket clusters without shafts). 6.7% of bristles on abdominal segments 3 and 4 are duplicated (2 hairs/2 sockets) and 11.6% show other defects. Homozygous clones in the notum lack most bristles and the few remaining ones have a four socket phenotype. 75% of the neurons and 50% of the sheath cells of the es organs are missing in phyl1/phyl2 embryos, although most es organs form.
Embryos derived from homozygous germline clones show defects dissimilar to those caused by defective tor signalling: head skeleton and filzkorper are abnormal.
Ommatidia of phyl2/phyl4 flies contain only 5 photoreceptor cells, an R8 and four cells of the R1-6 class. Rare ommatidia have five R1-R6 cells. This phenotype is also seen in clones of phyl2 cells. Missing receptor cells are transformed to cone cells.